Dissertation > Excellent graduate degree dissertation topics show
MPP~+-induced Autophagic Stress in Cellular Injury and Its Regulation Mechanisms
Author: ShiJiJun
Tutor: LiuChunFeng
School: Suzhou University
Course: Neurology
Keywords: Autophagy Autophagic stress α-synuclein MPP ~ (1 - methyl-4 - phenylpyridinium ion) Parkinson's disease Dynein
CLC: R736.6
Type: Master's thesis
Year: 2010
Downloads: 169
Quote: 0
Read: Download Dissertation
Abstract
|
The first part of the MPP ~ of MPP ~ autophagy induced stress in pheochromocytoma (PC12) cell damage induced PC12 cells to explore the purpose of self-macrophage stress and α-synuclein autophagic mechanism remove barriers, MPP ~ may lead to α-synuclein (α-synuclein) autophagic remove barriers to its abnormal accumulation mechanism and the relationship between the different levels of self-macrophage stress level of cell damage. Our group has successfully constructed over-expressing the human A30P pathogenic mutation of α-synuclein in PC12 cells model MPP ~ treated cells 24h after dimethylthiazol-salt (MTT) the detection cell viability, Western blot detection α-synuclein, micro tube associated protein 1 light chain 3 - Ⅱ (LC3-Ⅱ) and power proteins in the protein level of expression of the change and MDC staining and immune fluorescence labeled observe autophagy level of change and α-synuclein, LC3-Ⅱ and lysosomal-associated membrane protein -1 (LAMP-1) in the intracellular co-localization. At the same time, observed after treatment with different concentrations of MPP ~ the different autophagic stress levels of α-synuclein, LC3 and dynein expression at the protein level, and apoptosis. MPP ~ the viability of cells after treatment decreased significantly; the PC12 MPP ~ group (PC12 group: 0.200 ± 0.084; the A30P groups: 0.758 ± 0.085) compared with the untreated group (0.660 ± 0.072, P lt; 0.01) and A30P MPP to group (1.707 ± 0.400, P lt; 0.001) the expression of α-synuclein increased; LC3-II protein expression levels and the average number of autophagic vacuoles were increased, and the difference was statistically significant;, MPP to treatment of α- synuclein LC3-Ⅱ fluorescence signal and its colocalization increase, increase the the lysosomal fluorescence signal of the LAMP-1 marker, LC3-Ⅱ labeled autophagosome degree of co-localization was reduced. In addition, different concentrations of MPP ~ induced autophagy stress on PC12 cells have different effects on MPP ~ concentration increased autophagy stress levels were significantly higher increase in apoptotic cells, easy aggregation of α-synuclein protein increased expression mediated reduced from phages fusion with lysosomes key motor proteins - dynein expression. Conclusion MPP ~ autophagy induced stress impaired dynein autophagic remove barriers caused by α-synuclein is closely related to that dynein dysfunction plays an important role in cell damage, dynein α-synuclein self macrophages sexual degradation of the key targets of the regulation. The second part of the dynein: formation of autophagy stress, a key regulator of the target Objective To investigate the α-synuclein dynein-mediated degradation obstacles in the formation of autophagic stress effect and its mechanism, and provide new therapeutic strategies for the prevention and treatment of PD and-control targets. Constructed using siRNA knock-down of dynein cell model, then using immunocytochemistry and Western blot to observe changes in the expression of α-synuclein and autophagy levels, transmission electron microscopy and cell ultrastructural changes and autophagy morphological changes; intraperitoneal injection constructed rotenone-induced PD rat model, using the Western blot method rotenone-induced PD rat striatal dynein, α-synuclein and autophagy changes were observed and analyzed. Results of by siRNA technology interference power protein's function will cause of α-synuclein obvious aggregation (P lt; 0.01); rotenone treatment dynein expression decreased (P lt; 0.01), the expression of α-synuclein increased and aggregation (P lt; 0.01 increased), LC3-Ⅱ expression (since phages increase) (P lt; 0.05), of mTOR phosphorylation level decreased (ie inhibition of mTOR raised since macrophages) (P lt; 0.05), which showed that rotenone treatment caused power protein function obstacles The cause of α-synuclein aggregation increase as well as the increase in autophagosome formation (ie the autophagy stress level increased), mTOR involved in this process of autophagy induced. Conclusion dynein-mediated autophagic degradation of α-synuclein dysfunction mediated accumulation of α-synuclein aggregation and autophagy by autophagy dynein regulation of α-synuclein degradation potential adjustment in PD prevention targets.
|
Related Dissertations
- Effect of Different Glucose Concentrations on Autophagy in Neonatal Rat Cardiomyocyte Culture,R542.2
- Apoptosis Mechanisms Research in Interfering TIGAR Gene and Epirubicin Treatmnet in Hepatic Carcinoma Cells,R735.7
- The Effect of Gefitinib in Radiosensitization Through Autophagy Regulation in Human Non-small-cell Lung Cancer A549 Cell Line,R734.2
- Amiloride Is Protective in PC12 Cells Via Autophagy Pathway,R329
- The Effects of ER Stress on Cell Toxicity and Htt Levels in the Stable PC12 Cells Expressing Htt586,R363
- Intervention and Mechanisms of E-64d on Developmental Seizure-induced Cognitive Deficit,R720.597
- Effects of Lysosomal Enzyme Inhibitor on Recurrent Neonatal Seizure Induced Acute and Long-term Gene Expression and Behavioral Changes,R720.597
- The Effects of Autophagy on Cardiomyocytes Viability and Mitochondrial Membrane Potential in Sepsis,R459.7
- Autophagy marker protein LC3 lifetime studies in rats,R743
- Suppression of Autophagy-related Gene Causes Locomotive Defects in Drosophila,R741
- The Relationship between Phosphorylation of ataxin-3 and Autophagy,R744
- Power Frequency Magnetic Fields Induce Autophagy in the Chinese Hamster Lung Cells,R363
- Studies on the Cytoprotective Effect of Glycine and Its Mechanism,R965
- Detecting the Expression of the Autophagy-related Gene in the Nude Mouse Model of Human Lung Cancer Cell Line A549,R734.2
- Expression of Beclin-1 mRNA and Protein in Ovarian Endometriosis,R711.71
- The Expression of Osteopontin and Autophagy-related Genes in Abdominal Aortic Aneurysm and the Molecular Mechanism of Osteopontin-induced Autophagy of Smooth Muscle Cells,R543
- Melatonin Protects N2a Against Ischemia/Reperfusion Injury Through Autophagy Enhancement,R363
- Scavenger Receptor A Pathway Inhibits Endoplasmic Reticulum Stress-induced Autophagy in Macrophage,R730.2
- Study of Mechanisms of Jurkat and Raji Cell Death Induced by Resveratrol,R733.1
- The Changes and Significance of Autophagy after Subarachnoid Hemorrhage in Rats,R651.1
- Simulated Microgravity Induces Human Umbilical Vein Endothelial Cells Autophagy,R85
CLC: > Medicine, health > Oncology > Internal endocrine tumors > Adrenal tumors
© 2012 www.DissertationTopic.Net Mobile
|