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Effect of Nociceptive Electrical Stimulating Saphenous Nerve on Dopamine Content in Anterior Cingulate Gyrus of Rats and Its Mechanism

Author: LiuZhong
Tutor: HaoLiMing；WuMinFan
School: Jilin University
Course: Human Anatomy and Embryology
Keywords: Anterior cingulate gyrus Saphenous nerve Somatic pain Dopamine MK-801 CNQX High Performance Liquid Chromatography
CLC: R741
Type: Master's thesis
Year: 2011
Downloads: 19
Quote: 0
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Abstract


Objective To study the nociceptive saphenous nerve on cingulate its mechanism of dopamine content in front of the (ACG) . Method using high performance liquid chromatography - electrochemical detection technology research noxious electrical stimulation of the saphenous nerve (SN) at different times after the ACG dopamine content changes , as well as morphine , NMDA receptor antagonist MK -801 , AMPA / Kainate receptor antagonist CNQX dopamine content . Results noxious electrical stimulus SN for 15 min , ACG dopamine content significantly with increased , 30 min after increased most significantly , for 1 h after the start recovery , 2 h after the gradual recovery near control levels ; morphine inhibition of nociceptive electrical stimulus SN caused the ACG dopamine content the significantly higher . MK-801, CNQX antagonized the nociceptive SN caused ACG dopamine content was significantly higher. Conclusion This study proved nociceptive SN capable of cause ACG dopamine content in a time -dependent increased ; morphine inhibits nociceptive electrical stimulation of the the SN caused ACG dopamine content was significantly higher , suggesting the ACG accept SN incoming nociceptive information caused The functional activity of the ACG dopaminergic neurons enhanced dopaminergic system involved in the ACG the body pain sensation effect . MK-801, CNQX can antagonize nociceptive ACG SN caused increased dopamine content , suggesting that the NMDA receptor and AMPA / Kainate receptor involved in SN nociceptive information process incoming ACG increased dopamine content caused .

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