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Objective The topics to cerebral infarction and cerebral arteriosclerosis patients in this study, the joint neck and transcranial Doppler vascular ultrasound examination to explore the cerebral infarction and cerebral arteriosclerosis in patients with arterial disease differences exist for the early detection of cerebral infarction incidence of high-risk patients with cerebral arteriosclerosis basis. Methods meet diagnostic criteria for cerebral infarction in 57 patients aged 40 to 59 years old. Collected in line with domestic cerebral arteriosclerosis clinical symptoms and signs and by ultrasound examination confirmed the presence of cerebral arteriosclerosis in patients with 95 cases, aged 40 to 59 years old. Exclude polycythemia vera, diabetes, significant heart, liver dysfunction, atrial fibrillation, arteritis, cerebral hemorrhage, brain tumors, brain trauma, ultrasound can not penetrate the temporal occipital windows can not cope with sonographers. Collection period no significant positive signs of God neurological clinic within 29 cases as the control group, blood, blood sugar, blood lipids, blood pressure is normal; no electrocardiogram, retinal artery, neck and transcranial Doppler vascular ultrasound abnormalities. With a 2MHz pulsed Doppler probe by double temporal windows and windows were detected bilateral occipital cerebral artery, basilar artery, blood flow velocity record (Vd, Vd, Vm), pulsatility index (PI), resistance index (RI), S / D and spectral shape, respectively, the middle cerebral artery and basilar artery before and after as intracranial vascular representative. TCD spectrum in a single cardiac cycle selected spectrum to determine the start of each cardiac cycle (t0), the pulse weight notch point (ti) and the end point (te), while identifying the peak velocity point of time (tp), tp cardiac cycle starting point to calculate the time interval (Tp), starting from the time at which ti ventricular systolic (Ti), the starting point to the end point in the cardiac cycle at regular (T), calculated Tp ventricular systolic and the share of the entire cardiac cycle ratio (PTI-1, PTI-2). Color Doppler ultrasound probe with a 10MHz examination bilateral carotid artery and internal carotid arteries, the carotid bifurcation was measured at about 1.5cm proximal common carotid artery intima-media thickness (IMT), observe whether the plaques were measured plaque size, nature and number of records plaque area stenosis ratio measurement plaques (plaque thickness / lumen diameter). All data using SPSS13.0 software for statistical analysis. Results 1 compared with the control group: cerebral infarction group was significantly higher carotid IMT (P lt; 0.05), cerebral arteriosclerosis carotid IMT was no significant difference (P gt; 0.05); cerebral infarction and cerebral arteriosclerosis group middle cerebral artery and basilar artery PTI-1 and PTI-2 were significantly increased (P lt; 0.05). 2 group compared with cerebral arteriosclerosis: CI group fundus arteriosclerosis rate is not significantly higher (P gt; 0.05), myocardial ischemia was significantly higher (P lt; 0.05), carotid IMT was significantly thicker (P lt; 0.05), neck plaque formation was significantly higher (P lt; 0.05), plaque size integrals difference was significant (P lt; 0.05), stenosis ratio constitutes a difference was also statistically significant (P lt; 0.05) between the two groups plaques nature constitute no significant difference (P gt; 0.05). Cerebral infarction neck plaque formation accounted for 47.37%, a total of 44 patches, including 23 hard spots, soft plaque and mixed plaque 21, left 26, right 18. Neck cerebral arteriosclerosis group accounted for 27.37 percent plaque formation, a total of 40 patches, including morphea 18, soft plaque and mixed plaque 22, left 26, right 14. 3 group compared with cerebral arteriosclerosis: TCD spectrum morphological abnormalities of cerebral infarction group was significantly higher (P lt; 0.05), but the abnormal spectral type constitutes no significant difference (P gt; 0.05). Cerebral infarction peak ≥ S1 S2 peak (accounting for 69.35%); bimodal fusion (accounting for 28.23%); peaks after the extension (accounting for 2.42%). Peaks anterior circulation infarct group index PTI-1 and PTI-2 was significantly higher (P lt; 0.05), then the cycle peaks index PTI-1 and PTI-2 had no significant difference (P gt; 0.05). Between the two groups converged bimodal spectrum peaks index PTI-1 and PTI-2 were significantly different (P lt; 0.05). 4 cerebral arteriosclerosis arteriosclerosis group and cerebral infarction group were significantly different distributions (P lt; 0.05), cerebral infarction with carotid atherosclerosis with intracranial atherosclerosis dominated (43.86%), and cerebral arteriosclerosis dominated by intracranial atherosclerotic disease group (65.26%). 5.Logistic regression analysis: myocardial ischemia, medial thickness, diameter stenosis ratio of anterior circulation peaks Index PTI-1/PTI-2 and distributed in cerebral arteriosclerosis arteriosclerosis and cerebral infarction group OR values ??between groups were gt; 1, wherein the anterior circulation PTI-1/PTI-2 OR values ??were 2.57 and 1.99. Conclusion Myocardial ischemia, carotid intima-media thickness, diameter stenosis ratio of anterior circulation peaks and arteriosclerosis index PTI-1/PTI-2 distributed in cerebral arteriosclerosis and cerebral infarction differences between patients, suggesting that these factors may patients with cerebral infarction cerebral arteriosclerosis related.
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